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Vertigo (Recent Onset)

Population Covered By The Guidance

This pathway provides guidance on the imaging of adult patients with recent onset of vertigo.

Lead Researcher: Ci Yue Chia

Experts & Contributors: Daren Gibson, Jafri Kuthubutheen

Date reviewed: October 2022

Date Published: September 2025

Image 1a and 1b (Magnetic Resonance Imaging): Axial and sagittal images demonstrating a cavernous haemangioma in the left cerebellar hemisphere extending towards the vermis. Surrounding high signal intensity indicates oedema

Cavernous Haemangioma

Image 1a and 1b (Magnetic Resonance Imaging): Axial and sagittal images demonstrating a cavernous haemangioma in the left cerebellar hemisphere extending towards the vermis. Surrounding high signal intensity indicates oedema

Cavernous Haemangioma

  • Vertigo is a symptom of illusory movement arising from damage or dysfunction of the vestibular system.

  • A history and detailed neurological examination are important to determine whether the symptoms and signs are suggestive of a peripheral or central cause of vertigo. A focus towards identifying timing and triggers instead of dizziness type, has proven value in guiding imaging decisions; this is known as the timing, duration, triggers, and targeted examination (TiTrATE) diagnostic approach.

  • Head impulse, nystagmus, and test of skew (HINTS) testing for patients with acute onset spontaneous vertigo and nystagmus has high sensitivity and specificity for central cause.

  • Following specialist review and documentation of neurological findings, magnetic resonance imaging +/- magnetic resonance angiography (MRI +/- MRA) is generally indicated if a central cause of vertigo is suspected. 

  • If MRI is unavailable or significantly delayed, a computed tomography +/- angiography (CT +/- CTA) scan with fine images of the temporal bone/ posterior fossa is a reasonable substitute.

  • Vestibular migraine as per the Barany society definition needs to be considered in all cases of vertigo

Date of literature search: April 2022

References are graded from Level I to V according to the Oxford Centre for Evidence-Based Medicine, Levels of Evidence. Download the document

  1. Zwergal A, Dieterich M. Vertigo and dizziness in the emergency room. Curr Opin Neurol. 2020 Feb;33(1):117–25.
  2.  Kim SH, Park SH, Kim HJ, Kim JS. Isolated central vestibular syndrome. Ann N Y Acad Sci. 2015 Apr;1343(1):80–9.
  3.  An approach to vertigo in general practice. Australian Journal for General Practitioners [Internet]. 2016 Mar 22;45:190–4. Available from: https://www.racgp.org.au/afp/2016/april/an-approach-to-vertigo-in-general-practice
  4.  Choi JH, Park MG, Choi SY, Park KP, Baik SK, Kim JS, et al. Acute Transient Vestibular Syndrome. Stroke. 2017 Mar;48(3):556–62.
  5.  Guler A, Karbek Akarca F, Eraslan C, Tarhan C, Bilgen C, Kirazli T, et al. Clinical and video head impulse test in the diagnosis of posterior circulation stroke presenting as acute vestibular syndrome in the emergency department. Journal of Vestibular Research. 2017 Oct 21;27(4):233–42.
  6.  Lee SH, Kim JS. Differential diagnosis of acute vascular vertigo. Curr Opin Neurol [Internet]. 2020;33(1). Available from: https://journals.lww.com/co-neurology/Fulltext/2020/02000/Differential_diagnosis_of_acute_vascular_vertigo.22.aspx
  7.  Muncie HL, Sirmans SM, James E. Dizziness: Approach to Evaluation and Management. Am Fam Physician. 2017 Feb 1;95(3):154–62.
  8.  Spiegel R, Kirsch M, Rosin C, Rust H, Baumann T, Sutter R, et al. Dizziness in the emergency department: An update on diagnosis. Swiss medical weekly: official journal of the Swiss Society of Infectious Diseases, the Swiss Society of Internal Medicine, the Swiss Society of Pneumology. 2017 Dec 28;147.
  9.  Newman-Toker DE, Edlow JA. TiTrATE. Neurol Clin. 2015 Aug;33(3):577–99.
  10.  Edlow JA. Diagnosing Dizziness: We Are Teaching the Wrong Paradigm! Academic Emergency Medicine. 2013 Oct;20(10):1064–6.
  11.  Newman-Toker D, Curthoys I, Halmagyi G. Diagnosing Stroke in Acute Vertigo: The HINTS Family of Eye Movement Tests and the Future of the “Eye ECG.” Semin Neurol. 2015 Oct 6;35(05):506–21.
  12.  Tarnutzer AA, Berkowitz AL, Robinson KA, Hsieh YH, Newman-Toker DE. Does my dizzy patient have a stroke? A systematic review of bedside diagnosis in acute vestibular syndrome. Can Med Assoc J. 2011 Jun 14;183(9):E571–92.
  13.  Newman-Toker DE, Kerber KA, Hsieh YH, Pula JH, Omron R, Saber Tehrani AS, et al. HINTS Outperforms ABCD2 to Screen for Stroke in Acute Continuous Vertigo and Dizziness. Academic Emergency Medicine. 2013 Oct;20(10):986–96.
  14.  Saber Tehrani AS, Kattah JC, Mantokoudis G, Pula JH, Nair D, Blitz A, et al. Small strokes causing severe vertigo: Frequency of false-negative MRIs and nonlacunar mechanisms. Neurology. 2014 Jul 8;83(2):169–73.
  15.  Batuecas-Caletrío Á, Yáñez-González R, Sánchez-Blanco C, González-Sánchez E, Benito J, Gómez JC, et al. [Peripheral vertigo versus central vertigo. Application of the HINTS protocol]. Rev Neurol. 2014 Oct 16;59(8):349–53.
  16.  Chen L, Lee W, Chambers BR, Dewey HM. Diagnostic accuracy of acute vestibular syndrome at the bedside in a stroke unit. J Neurol. 2011 May 12;258(5):855–61.
  17.  Huh YE, Koo JW, Lee H, Kim JS. Head-Shaking Aids in the Diagnosis of Acute Audiovestibular Loss due to Anterior Inferior Cerebellar Artery Infarction. Audiology and Neurotology. 2013;18(2):114–24.
  18.  Cohn B. Can Bedside Oculomotor (HINTS) Testing Differentiate Central From Peripheral Causes of Vertigo? Ann Emerg Med. 2014 Sep;64(3):265–8.
  19.  Kerber KA, Newman-Toker DE. Misdiagnosing Dizzy Patients. Neurol Clin. 2015 Aug;33(3):565–75.
  20.  Kattah JC, Talkad A v., Wang DZ, Hsieh YH, Newman-Toker DE. HINTS to Diagnose Stroke in the Acute Vestibular Syndrome. Stroke. 2009 Nov;40(11):3504–10.
  21.  Chen R, Su R, Deng M, Liu J, Hu Q, Song Z. A Posterior Circulation Ischemia Risk Score System to Assist the Diagnosis of Dizziness. Journal of Stroke and Cerebrovascular Diseases. 2018 Feb;27(2):506–12.
  22.  Navi BB, Kamel H, Shah MP, Grossman AW, Wong C, Poisson SN, et al. Application of the ABCD2 Score to Identify Cerebrovascular Causes of Dizziness in the Emergency Department. Stroke. 2012 Jun;43(6):1484–9.
  23.  Kuroda R, Nakada T, Ojima T, Serizawa M, Imai N, Yagi N, et al. The TriAGe+ Score for Vertigo or Dizziness: A Diagnostic Model for Stroke in the Emergency Department. J Stroke Cerebrovasc Dis. 2017;26 5:1144–53.
  24.  Paul NL, Simoni M, Rothwell PM. Transient isolated brainstem symptoms preceding posterior circulation stroke: a population-based study. Lancet Neurol. 2013 Jan;12(1):65–71.
  25.  Hoshino T, Nagao T, Mizuno S, Shimizu S, Uchiyama S. Transient neurological attack before vertebrobasilar stroke. J Neurol Sci. 2013 Feb;325(1–2):39–42.
  26.  Compter A, Kappelle LJ, Algra A, van der Worp HB. Nonfocal Symptoms are More Frequent in Patients with Vertebral Artery than Carotid Artery Stenosis. Cerebrovascular Diseases. 2013;35(4):378–84.
  27.   Kerber KA, Brown DL, Lisabeth LD, Smith MA, Morgenstern LB. Stroke Among Patients With Dizziness, Vertigo, and Imbalance in the Emergency Department. Stroke. 2006 Oct;37(10):2484–7.
  28.  Kim AS, Fullerton HJ, Johnston SC. Risk of Vascular Events in Emergency Department Patients Discharged Home With Diagnosis of Dizziness or Vertigo. Ann Emerg Med. 2011 Jan;57(1):34–41.
  29.  Lee CC, Ho HC, Su YC, Chiu BCH, Su YC, Lee YD, et al. Increased Risk of Vascular Events in Emergency Room Patients Discharged Home with Diagnosis of Dizziness or Vertigo: A 3-Year Follow-Up Study. PLoS One. 2012 Apr 27;7(4):e35923.
  30.  Newman-Toker DE, Saber Tehrani AS, Mantokoudis G, Pula JH, Guede CI, Kerber KA, et al. Quantitative Video-Oculography to Help Diagnose Stroke in Acute Vertigo and Dizziness. Stroke. 2013 Apr;44(4):1158–61.
  31.  Chalela JA, Kidwell CS, Nentwich LM, Luby M, Butman JA, Demchuk AM, et al. Magnetic resonance imaging and computed tomography in emergency assessment of patients with suspected acute stroke: a prospective comparison. The Lancet. 2007 Jan;369(9558):293–8.
  32.  Ozono Y, Kitahara T, Fukushima M, Michiba T, Imai R, Tomiyama Y, et al. Differential diagnosis of vertigo and dizziness in the emergency department. Acta Otolaryngol. 2014 Feb 6;134(2):140–5.
  33.  Saber Tehrani AS, Coughlan D, Hsieh YH, Mantokoudis G, Korley FK, Kerber KA, et al. Rising Annual Costs of Dizziness Presentations to U.S. Emergency Departments. Academic Emergency Medicine. 2013 Jul;20(7):689–96.
  34.  Choi SY, Kim HJ, Kim JS. Chasing dizzy chimera: Diagnosis of combined peripheral and central vestibulopathy. J Neurol Sci. 2016 Dec;371:69–78.
  35.  Murakami T, Nakayasu H, Doi M, Fukada Y, Hayashi M, Suzuki T, et al. Anterior and posterior inferior cerebellar artery infarction with sudden deafness and vertigo. Journal of Clinical Neuroscience. 2006 Dec;13(10):1051–4.
  36.  Connor SEJ, Sriskandan N. Imaging of dizziness. Clin Radiol. 2014 Feb;69(2):111–22.
  37.  Sparaco M, Ciolli L, Zini A. Posterior circulation ischemic stroke—a review part II: imaging and acute treatment. Neurological Sciences. 2019 Oct 24;40(10):2007–15.
  38.  Zhang XH, Liang HM. Systematic review with network meta-analysis. Medicine. 2019 Jul;98(30):e16360.
  39.  Morita S, Suzuki M, Iizuka K. False-negative diffusion-weighted MRI in acute cerebellar stroke. Auris Nasus Larynx. 2011 Oct;38(5):577–82.
  40.  Juliano AF. Cross Sectional Imaging of the Ear and Temporal Bone. Head Neck Pathol. 2018 Sep 1;12(3):302–20.
  41.  Shama SA, Eid M, Mehanna AMA, Eissa LA. Dehiscences of the semicircular canals as discrete third window lesions of the inner ear. The Egyptian Journal of Radiology and Nuclear Medicine. 2013 Mar;44(1):15–21.
  42.  Machner B, Erber K, Choi JH, Trillenberg P, Sprenger A, Helmchen C. Usability of the head impulse test in routine clinical practice in the emergency department to differentiate vestibular neuritis from stroke. European Journal of Neurology. 2021 May;28(5):1737-44.

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ACUTE VERTIGO History and Examination TiTrATE algorithm +/- HINTS+ if appropriate MRI IAMs + Brain + Post Gadolinium imaging Consistent with peripheral aetiology Normal neurological examination, abnormal HIT, fixed (unidirectional) horizontal nystagmus OR mixed (not pure) torsional and vertical nystagmus, no skew deviation Consistent with central aetiology Presence of risk factors for stroke/vascular disease, neurological signs or severe headache, atypical nystagmus, ataxia, skew deviation present Uncertain aetiology Suspected SSC dehiscence/noise-induced vertigo/history of trauma or surgery/chronic otitis media (with ear pain, hearing loss, facial weakness or ear discharge) Suspected BPPV/Meniere’s/viral neuronitis or labyrinthitis (incl. Ramsay Hunt Syndrome) Without any atypical features With unilateral or asymmetrical audiological Sx Appropriate Management +/- ENT referral Appropriate emergent Mx +/- ENT opinion Is presentation acute/sudden onset (e.g., stroke, head trauma, etc)? Yes No Consider vestibular migraine Neurology referral Appropriate emergent Mx +/- neurology opinion Appropriate emergent Mx +/- neurology opinion Is stroke themost likely DDx? Yes No Appropriate emergent Mx +/- neurology opinion MRI may still be necessary to exclude a central cause Low risk (e.g., ABCD2 ≤ 3, PCI = 0) Moderate to High (e.g., ABCD2 >3, PCI >0) Atypical presentation?(e.g., unsteadiness, unilateral hearing loss, noise induced dizziness, s-AVS, complex nystagmus, etc) Investigate as per suggestive of central aetiology Yes No Abbreviations Key BPPV: Benign Paroxysmal Positional VertigoCISS: Constructive Interference in Steady State (a gradient echo MRI sequence) HINTS+: Head Impulse, Nystagmus, Test of Skew + hearing loss examinationHIT: Head Impulse TestIAM: Internal Auditory MeatusPCI: Posterior Circulation Ischemia risk score s-AVS: spontaneous Acute Vestibular Syndrome (a classification within TiTrATE tool)SSC: Superior Semicircular Canal TiTrATE: Timing, Duration, Triggers and Target Examination diagnostic tool Appropriate emergent Mx +/- ENT opinion MRI + MR Angiography (MRA) of the brain + CISS IAMs MRI + MR Angiography (MRA) of the brain CT Head +/- contrast with thin slices through posterior fossa(+/- CT temporal bone if Hx of trauma, otorrhoea or prior middle ear/mastoid surgery) Assess stroke risk Clinical follow-up CT petrous bones Refer to stroke protocol

History and Examination

A focus towards identifying timing (including speed of symptom onset), duration, description of the vertigo and triggers instead of dizziness type, has proven value in guiding imaging decisions. HINTs+ (head impulse, nystagmus, test of skew + hearing loss) is a relatively simple bedside test that can be used to rule out a central cause. HINTs+ assesses head impulse, nystagmus and test of skew with assessment of cranial nerves & includes hearing loss.

  • History and detailed neurological examination are important in distinguishing between central and peripheral aetiology and will inform subsequent diagnostic evaluation and treatment. Diagnostic index tests and risk stratification tools like TiTrATE (symptom timing, triggers, and targeted bedside eye examinations), HINTS+ (Head Impulse, Nystagmus, Test of Skew + hearing loss), ABCD2 (age, blood pressure, clinical features, duration of symptoms, and diabetes) are useful in estimating the risk of central pathology and assessing stroke risk .

  • A large body of recent literature suggests a change of paradigm in how to assess patients with vertigo or dizziness, by identifying timing and triggers (TiTrATE diagnostic approach ) instead of type of dizziness to optimise usage of targeted bedside examinations of proven value .

    • Patients have difficulty describing quality of their symptoms – with 50% changing their dizziness type – but more consistently identify timing and triggers .

  • HINTS has demonstrated superiority over vascular risk factor stratification (ABCD2 score) and continued superiority over diffusion weighted magnetic resonance imaging (DW-MRI) , particularly in patients with smaller strokes . Similar findings have been confirmed by other groups and two systematic reviews . 

    • This requires correct identification of acute vestibular syndrome (AVS) patients  

    • Acute vestibular syndrome (AVS) is characterized by the rapid onset (over seconds to hours) of vertigo, nausea/vomiting, and gait unsteadiness in association with head-motion intolerance and nystagmus lasting days to weeks .

    • The head impulse test (HIT) should be reassessed by a neuro-otological expert or quantitative video-oculography HIT to improve accuracy. Currently, accuracy of bedside HIT tests when performed by non-experts is only 58%, with a high sensitivity of 88% but low specificity of 64% for peripheral vertigo .

  • HINTS+ risk stratification tool includes additional consideration of hearing loss in risk stratification. Clinically, hearing loss is a useful finding in guiding further imaging in the Emergency Department.

    • An audiology review prior to semi-urgent MRI may be considered to confirm hearing loss, especially if aetiology is uncertain. 

    • Tinnitus, unilateral or asymmetrical hearing loss may also prompt further investigation with semi-urgent MRI.

Assess Stroke Risk

Important to exclude stroke as cause of dizziness, ensure to screen appropriately regardless of age.

  • When uncertain of central or peripheral aetiology, stroke risk should be assessed in all ages using clinically validated stroke risk scales such as ABCD2 and posterior circulation ischemia (PCI) risk score. 

  • The PCI risk score could help clinicians differentiate patients with a PCI rapidly, with a sensitivity of 94.1% and a specificity of 41.4% .

  • ABCD2 risk score (age, blood pressure, clinical features, diabetes) has been shown to have some utility in determining the risk of stroke in ED patients presenting with AVS . An ABCD2 score cut off >3 for diagnosing stroke in AVS has a sensitivity ranging from 61.1-86.3% and specificity ranging from 58.3-62.3%..

  • The ABCD2 score contributes to the diagnosis of vestibular stroke , but is less sensitive compared to the PCI risk score .

Acute Vertigo

Vertigo is a symptom of illusory movement in the absence of physical movement arising from damage or dysfunction of the sensory vestibular pathway. “Dizziness” is a term that is less preferable and should be avoided. Consider using the term “imbalance” or “disequilibrium”. Lightheadedness is usually not equivalent to vertigo and other causes should be considered.

  • Vertigo and dizziness are a common emergent presentation and mostly related to peripheral (end organ vestibular) causes which are generally benign. Approximately 25% are due to central causes (e.g., posterior fossa haemorrhage, tumour, vessel dissection, atheroma, or infarction) which, if missed, could lead to adverse morbidity and mortality .

  • A spontaneous, acute onset of vestibular symptoms increases the risk of stroke 3 to 5-fold .

  • Posterior circulation stokes are at especially high risk of misdiagnosis, 24-60% of patients often present with mild, non-specific, or transient symptoms .

  • Stroke risk in patients presenting isolated spontaneous acute vestibular syndrome (AVS) without vascular risk factors is around 10–20%, and 25% of strokes occur in patients under the age 50 . 

  • This cautions against only imaging older patients with vascular risk factors, as overreliance on youth, low vascular risk, normal neurologic exam and normal CT would likely explains the relatively high odds of missed stroke in isolated dizziness .

  • In the future, quantitative Head Impulse, Nystagmus, Test of Skew + hearing loss (HINTS) testing might be used in the emergency department (ED) to discharge AVS patients unlikely to have a stroke without any complex, onward imaging, accelerate access to acute therapies without awaiting an out-patient MRI study.

Computed Tomography (CT) of the Head

Head CT & acute stroke protocols (Non-Enhanced CT, CT Angiography, CT perfusion; NECT, CTA, CTP) are frequently used in emergent presentations where timely MRI is not available. Caution that NECT Brain may miss subtle or early brainstem and cerebellar ischaemia/infarcts.

  • Head CT is frequently used in emergent presentations where magnetic resonance imaging (MRI) is not available, impractical, or contraindicated to exclude posterior fossa haemorrhage or a large mass as a cause for vertigo .

  • It is considerably less sensitive in detecting posterior fossa, brainstem pathology and an ischemic stroke compared to diffusion weighted magnetic resonance imaging (DW-MRI), prospective comparison of CT with MRI showed CT has a very low sensitivity (maximum 16% compared with 83% for DW-MRI) to identify acute ischaemic stroke , particularly within posterior fossa (7%).

  • Fine cuts through the cerebellum should be used to assist with diagnosis (1,31). If immediate brain imaging is indicated and a non-enhanced CT +/- CT angiography/CT perfusion (NECT +/-CTA/CTP) is obtained at presentation, subsequent MRI and magnetic resonance angiography (MRA) are generally recommended post 48 hours . In the meantime, the patient's neurological status should be closely monitored .

Magnetic Resonance Imaging (MRI) of the Internal Auditory Meatus (IAM) and the Brain + Constructive Interference in Steady State (CISS) sequence of the Internal Auditory Meatus (IAMs)

To exclude tumours of the cerebellopontine angle cistern and internal auditory meatus. This should be routinely requested if an MRI scan of the brain is to be performed for the assessment of vertigo to exclude cochlear and retro cochlear pathology.

  • Investigation of choice after clinical examination if:

    • A central cause for vertigo is suspected (e.g., vertebro-basilar infarction, intracranial haemorrhage, brainstem neoplasm, multiple sclerosis, or infection) .

    • A central/peripheral distinction cannot be made, and patient has moderate to high stroke risk or an atypical presentation (e.g., unsteadiness, unilateral hearing loss, noise induced dizziness, spontaneous acute vestibular syndrome (AVS), suspected subarachnoid haemorrhage or brainstem encephalitis) .

  • MRI has a higher diagnostic yield than CT in detecting central structural causes of vertigo

  • MRA has been shown to have a sensitivity of 97% and specificity of 98.9% for the diagnosis of large vessel occlusions and stenoses of the posterior circulation when compared to the reference standard of intra-arterial angiography .

  • A systematic review with meta-analysis demonstrated that diffusion weighted imaging (DWI) has a high sensitivity & specificity for acute posterior circulation infarction when compared with traditional CT, CT angiography (CTA), MRI and MR angiography (MRA) .

  • Caution is advised in patients with acute vestibular syndrome and suspected ischemic stroke. Be cautious of an early, negative DW-MRI within the first 48 hours as DW-MRI misses 10-20% of larger strokes and up to 50% of small (<1cm) brainstem and cerebellar strokes with a <1 cm diameter .

  • Repeat delayed DW-MRI (3–7 days after onset of symptoms) may be required to confirm a new infarct .

  • MRI (+/- non-echo-planar DWI if suspected cholesteatoma) can be used to evaluate the extent of peripheral labyrinthine or intracranial involvement .

  • A prospective study showed that perfusion-weighted imaging (PWI) may contribute to identification of ischemic strokes especially in those with initially negative DWI .

Magnetic Resonance Imaging (MRI) of the Internal Auditory Meatus (IAM) and the Brain + Post Gadolinium imaging

MRI has a higher diagnostic yield than CT in detecting central and peripheral causes of vertigo. Post Gadolinium imaging is routine in diagnosing Meniere’s, viral neuronitis/labyrinthitis unless contraindicated.

  • If vertigo is associated with unilateral sensorineural hearing loss, MRI of the internal auditory meatus and temporal bone and brain are recommended as to exclude tumours of the cerebellopontine angle cistern, internal auditory meatus, cochlear and brainstem, most commonly an acoustic neuroma (.

  • Additional audiological testing is also recommended .

  • High resolution T2w isovolumetric imaging is routine. 

  • Post Gadolinium imaging is routine in Meniere’s and viral neuronitis/labyrinthitis unless contraindicated.

Computed Tomography (CT) of Petrous Bones (high resolution)

Investigation of choice in cases of suspected acute otitis media/chronic suppurative otitis media (AOM/CSOM), barotrauma, superior semicircular canal dehiscence, cholesteatoma, and alternative diagnoses.

  • Non-enhanced CT (NECT) of petrous bones is the confirmatory diagnostic investigation of choice in cases of suspected superior semicircular canal (SSC) dehiscence, third-window phenomena or other middle ear/ skull base erosive pathology (history of trauma/surgery/chronic suppurative otitis media and progressive hearing loss) .

  • High resolution CT with thin slices through the petrous temporal bone may demonstrate a defect in the bony covering (arcuate eminence) of the SSC, best appreciated in the coronal plane . 

  • With compromised radiation doses, ultrathin slices, high resolution CT petrous bone scans can achieve a sensitivity of 100% and a specificity of 99% in the identification of superior SSC dehiscence . 

  • Contrast is rarely required.

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    • Production
      • Initial Engagement with Consumers
      • Principles for Creating and Managing Content